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Can environmental pollution increase the risk of cancer?
Yes ,it is right to say that the environmental pollution can increase the risk of cancer. As the environmental pollution is increasing day-to-day is causing bad impact on our health. As the pollution increases our immune system or stamina of normal being is decrease . Which will decrease our body stRead more
Yes ,it is right to say that the environmental pollution can increase the risk of cancer. As the environmental pollution is increasing day-to-day is causing bad impact on our health. As the pollution increases our immune system or stamina of normal being is decrease . Which will decrease our body stamina to revert back against the viruses,bacteria,disease,etc. and mack as sick. In our daily life we inhale the air and this can damage our lungs ,because air pollution contains a mixture of polluting particles, there are a few ways air pollution could cause lung cancer. For example, tiny particles may build up in the lungs and damage the DNA in cells. This can change how cells divide, which can lead to cancer. Another example of this is the depletion of ozone layer which is the major effect of pollution. The ozone depletion is not only an effect but as well as a concerning issue to discus. As the ozone layer is depleting the harmful UV raise or ultraviolet radiation will directly enter in the earth’s boundary which will harm to our skin and also cause cancer. So , we have not only protect our environment or earth but also ourselves from the disease.
Thank you!
See lessHow do specific genetic mutations contribute to the development of diseases like cancer or Alzheimer’s?
Genetic mutations play a pivotal role in the development of diseases like cancer and Alzheimer’s by altering normal cellular functions. In cancer, mutations can occur in genes that regulate cell growth, division, and apoptosis, leading to uncontrolled cell proliferation. Oncogenes, when mutated, becRead more
Genetic mutations play a pivotal role in the development of diseases like cancer and Alzheimer’s by altering normal cellular functions. In cancer, mutations can occur in genes that regulate cell growth, division, and apoptosis, leading to uncontrolled cell proliferation. Oncogenes, when mutated, become overactive and promote tumor growth, while tumor suppressor genes lose their ability to control cell division and repair DNA damage. For instance, mutations in the TP53 gene, which encodes the p53 protein, impair its function as a tumor suppressor, leading to unchecked cellular growth and cancer progression.
In Alzheimer’s disease, genetic mutations can disrupt neuronal function and promote the accumulation of toxic proteins. Mutations in the APP, PSEN1, and PSEN2 genes are associated with early-onset Alzheimer’s. These mutations result in the abnormal processing of amyloid precursor protein (APP), leading to the accumulation of amyloid-beta plaques, a hallmark of Alzheimer’s pathology. Additionally, mutations in the APOE gene, particularly the APOE ε4 allele, increase the risk of late-onset Alzheimer’s by influencing amyloid-beta deposition and clearance, as well as lipid metabolism and neuronal repair.
These genetic alterations, through their impact on cellular pathways, contribute significantly to the onset and progression of complex diseases like cancer and Alzheimer’s.
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